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These perturbations are partly because of dependency on p53 [13]. This review displays that MIF serves a task in nervous process development, Even though the correct character of this role remains undefined.
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that's, stimulation of CD74 with exogenous MIF could cause an abnormal inflammatory state, as MIF is capable of stimulating CXCR2 and CXCR4 receptors In combination with CD74. CXCR2 click here and CXCR4 receptors when activated encourage inflow of neutrophils and lymphocytes, respectively (63, sixty four).
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MIF activates the proliferation and differentiation of neural stem and progenitor cells (NSPC) in the nicely-recognized Wnt/β-catenin signal pathway (Figure one). NSPCs are self-renewing cells which might be responsible for The expansion and progress of anxious tissue in conjunction with neural plasticity.
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